Thank you for writing on substack. I wish I could understand the details of all of what you have written :)! I would second the request for you to weigh in on the micro clotting issue. We are seeing a lot of ppl post C19 with dyspnea; failing six minute walks etc. even after decent treatment regimes. Some are gradually getting better, others struggle still. As a nurse, it feels like I am in a slow motion film and the population is slowly being disabled before my eyes. Looking for pathways of healing for these folks.
The basis of SARS-2 mortality is blocking interferon-1 which unregulated HERV-K102, in the absence of which causes ADE from infected monocytes and neutrophils. Wow! Isn’t nature ironical to use viruses to kill viral infections. Perfect!
We are trying to understand the mechanisms of interaction occurring to create these rubbery proteins, clots, that they (embalmers) are pulling out of vaccinated (only vaccinated) bodies. Why are they triggering amyloid proteins within the circulatory system? Any insights would be appreciated. Thank you for being heroic.
Please see a relevant article on the procoagulant property of S protein by Ryu JK et al., bioRxiv, Oct 13, 2021. Also see Barberis E et al Front Mol Biosci Feb 2021 where the proteins of exosomes in plasma from critical and non-critical C-19 patients are compared. A2M is a protease inhibitor implicated in Alzheimer's disease (AD) where it degrades and clears the A-beta. The S protein of SC2 interacts with A2M (Chen Z et al., EMBO J Sept 1 2021) and A2M was elevated in the exosomes from the critical C-19 patients as part of the hypercoagulatory state. APOE and APP (the beta amyloid precursor) are implicated in AD and in SARS-CoV-2 infection of the monocytes and macrophages. SC2 may downregulate A2M in these myeloid cells. Since both APOE and APP are in the ivermectin -SC2 PPI network, presumably ivermectin would be expected to reverse the hypercoagulation and prevent the odd clots as may be found in people who died following the vaccine. I hope this helps.
In response to your request I have added information by Sweeney et al with respect to the inflammatory or hypercoagulation category and risk of death in COVID-19 patients. I don't think I can top Dr. Ryan Cole's expertise on the subject, given that he has examined these clots under the microscope. We might be seeing more of these in deceased COVID-19 patients due to a more prominent downregulation of DDX6 related to higher levels of ADE related to omicron infection mediated death. Please see previous blog on the unexpected higher incidence of death due to omicron and how in slow to vaccinate nations, natural infection by delta seems to have provided better protection against omicron mediated death.
Thank you for writing on substack. I wish I could understand the details of all of what you have written :)! I would second the request for you to weigh in on the micro clotting issue. We are seeing a lot of ppl post C19 with dyspnea; failing six minute walks etc. even after decent treatment regimes. Some are gradually getting better, others struggle still. As a nurse, it feels like I am in a slow motion film and the population is slowly being disabled before my eyes. Looking for pathways of healing for these folks.
See https://phmpt.org/wp-content/uploads/2021/11/5.3.6-postmarketing-experience.pdf pages 23 and 24 for PFIZER data relating to blood clotting serious side effects of vaccine.
Please see the two comments to the BarefootHealer for update.
The basis of SARS-2 mortality is blocking interferon-1 which unregulated HERV-K102, in the absence of which causes ADE from infected monocytes and neutrophils. Wow! Isn’t nature ironical to use viruses to kill viral infections. Perfect!
Reminds me of the old adage to fight fire with fire!
Read about how humans only recently acquired this protector system against emerging RNA viruses (epidemic/pandemic) in my first post on Substack.
👏👏👏👏❤.
If it's not too much trouble, could you also please turn your hand at the clotting issue occurring post vaccination, that the mortician and embalmers have brought to Dr Ryan Cole, pathologist, as shown in Steve Kirsch rumble video. https://rumble.com/vxeqvu-pathologist-on-ryan-cole-on-the-mystery-blood-clots.html
We are trying to understand the mechanisms of interaction occurring to create these rubbery proteins, clots, that they (embalmers) are pulling out of vaccinated (only vaccinated) bodies. Why are they triggering amyloid proteins within the circulatory system? Any insights would be appreciated. Thank you for being heroic.
Please see a relevant article on the procoagulant property of S protein by Ryu JK et al., bioRxiv, Oct 13, 2021. Also see Barberis E et al Front Mol Biosci Feb 2021 where the proteins of exosomes in plasma from critical and non-critical C-19 patients are compared. A2M is a protease inhibitor implicated in Alzheimer's disease (AD) where it degrades and clears the A-beta. The S protein of SC2 interacts with A2M (Chen Z et al., EMBO J Sept 1 2021) and A2M was elevated in the exosomes from the critical C-19 patients as part of the hypercoagulatory state. APOE and APP (the beta amyloid precursor) are implicated in AD and in SARS-CoV-2 infection of the monocytes and macrophages. SC2 may downregulate A2M in these myeloid cells. Since both APOE and APP are in the ivermectin -SC2 PPI network, presumably ivermectin would be expected to reverse the hypercoagulation and prevent the odd clots as may be found in people who died following the vaccine. I hope this helps.
In response to your request I have added information by Sweeney et al with respect to the inflammatory or hypercoagulation category and risk of death in COVID-19 patients. I don't think I can top Dr. Ryan Cole's expertise on the subject, given that he has examined these clots under the microscope. We might be seeing more of these in deceased COVID-19 patients due to a more prominent downregulation of DDX6 related to higher levels of ADE related to omicron infection mediated death. Please see previous blog on the unexpected higher incidence of death due to omicron and how in slow to vaccinate nations, natural infection by delta seems to have provided better protection against omicron mediated death.